FREQUENTLY ASKED QUESTIONS 123
What are the most commonly used BFRs and in
what products are they used?
The two most commonly used BFRs are Tetrabromobisphenol A (TBBPA) and Decabromodiphenyl ether (Deca-BDE). These 2 products make up approximately 50% of all BFRs produced and used. TBBPA is the main flame retardant used in circuit boards in computers and other electronic products. TBBPA is also used in the housings of computer monitors. Deca-BDE is commonly used to flame retard plastics in electrical and electronic equipment such as television set casings and the insulation around wire and cable.
What are some of the other products that use
BFRs as flame retardants?
The foams for thermal insulation used in building and home construction
are typically flame retarded with Hexabromocyclododecane (HBCD).
Upholstered fabrics for commercial furniture, cars, and residential
furniture in some parts of the world are flame retarded with either
Deca-BDE or HBCD. Some roofing materials and coatings are also
flame retarded with a BFR.
Is my TV or computer exposing me or my family
to BFRs?
It is highly unlikely. The plastics used in TV and computer housings are hard and dense so that substances used in their construction have little opportunity to be released. Also, the BFRs commonly used in these applications do not tend to move from their solid state into a gas form, and so are unlikely to be released into the air around the computer or TV. Monitoring studies have confirmed that TVs and computers are not significant emission sources.
Chemicals called PBDEs have been in the
news lately. What are they?
The term PBDEs is an abbreviation for polybrominated diphenyl
ethers. Another term for these chemicals is polybrominated diphenyl
oxides. PBDEs represent one particular family of BFRs. There are
three commercial PBDE products (Octa-BDE, Penta-BDE and Deca-BDE)
and the three products differ in their composition, uses, manufactured
volume, toxicology, and environmental distribution. Of the three
PBDEs, only Deca-BDE remains in widespread commercial use. Because
of this, we believe that rather than referring to all PBDEs as
if they all had the same properties, it is preferable to state
the specific commercial product or the specific identity of the
molecule.
What PBDEs have been found in breast milk?
Are the levels going up, down or staying the same in various regions
around the world?
The PBDEs detected in breast milk are similar to those detected
in fish caught in the wild. About 50-70% of the total amount is
commonly made up of one specific molecule: 2,2’,4,4’-TetraBDE.
The next most common is 2,2’,4,4’,5-Penta-BDE. These
specific molecules are major components of one PBDE product: “Penta-BDE”.
It’s been reported that total PBDE levels in breast milk
collected in Sweden increased during the 1990s’, and peaked
in 1997, but this is based on a small number of samples. Time trend
data doesn’t exist for other countries in Europe, in the
U.S. or Asia. Penta-BDE has since been removed from markets and
detection levels are expected to decrease.
How did these compounds get in breast milk?
At this point, we can’t answer this question with any degree of certainty. There simply has not been enough research to positively identify how the women in the studies were exposed. For other environmental compounds, diet is typically the major source. Recent work measuring levels in a typical “market basket” tends to indicate this is also true with regard to “PBDEs”, but at this point it is too early to be certain.
Do the levels in breast milk pose a risk to
babies?
Our best information indicates that the levels found in breast milk are hundreds or thousands of times below that which might cause a harmful effect. Leading authorities in the field continue to recommend breast milk as the best source of nutrition for babies.
A number of publications report detecting various
BFRs in sediments, fish, birds and other species. What do the levels
reported mean?
BFRs detected in the environment include Deca-BDE, TBBPA, HBCD, and various tetra-, penta-, and hexa-BDEs. The amounts and locations of each vary, and depend on the molecule’s properties and the products use. For example, when Deca-BDE is detected, it is usually in sediments near point sources of manufacture or industrial use. This is because Deca-BDE is either not taken up at all by animals, or taken up very sparingly and so does not tend to move in the environment far from its point of release. HBCD, on the other hand, is taken up more efficiently and has been detected in fish and birds. Generally, levels in environmental samples are very low and in the parts-per-billion or parts-per-trillion range.
There’s a lot of speculation in the news
about the kind of health or environmental effects that could be
caused by BFRs. What effects have actually been found in humans
or in animals in the environment?
It’s easy to raise questions in the media and speculate on
what might happen. Those raising questions aren’t required
to have any particular expertise or to have demonstrated knowledge
about the existing database. To date, no human health or environmental
effects have been associated with the BFRs detected.
Are BFRs persistent in the environment?
Some BFRs seem to be persistent; others do not. For example, Deca-BDE
seems resistant to degradation in sediments, but studies have shown
that both TBBPA and HBCD can be degraded in sediments and/or soils.
Other BFRs have not been studied to the extent of these products.
Do BFRs accumulate in tissues?
There seems to be a common misperception that BFRs, as a group,
accumulate in animal and human tissues. However, studies have shown
that this is not the case. Neither Deca-BDE nor TBBPA, which make
up 50% of all BFRs sold, accumulate.
Do BFRs affect the endocrine system of humans
or animals?
The endocrine system has become an active area of toxicology research
in recent years. It is composed of several organs: pituitary, thyroid,
parathyroid, pancreas, adrenals, ovaries and testicles, all of
which produce hormones that act on other parts of the body to integrate
their various functions. No BFR has been shown to affect the endocrine
system of humans. Changes in thyroid hormone blood levels in response
to the Penta-BDE , HBCD and TBBPA have been shown in rats. However,
there was no associated harmful effect in the whole animal, and
the mechanism by which the thyroid hormone level was lowered is
not believed applicable to humans.
Are BFRs toxic to the nervous system?
Exposure to large, repeated doses of Deca-BDE, TBBPA, or HBCD
over either the entire or a substantial portion of the animals’ lifetime
has had no effect on their nervous systems. Similarly, no effect
on the fetal nervous system was seen when the pregnant females
were treated. One group of researchers has reported that 2,2’,4,4’-Tetra-BDE,
2,2’,4,4’,5-Penta-BDE and Deca-BDE can cause behavioral
or learning effects when given to 10 or 3 day old mice. Neither
mice nor the study design are those typically used to study neurotoxicity.
Further, the reported effects could not be reproduced in rats.
Why do people use the term BFRs when the individual
BFRs are so different from one another? Can you really talk about
them as one homogeneous group?
It is really not correct to imply that all BFRs are alike, because
they come in many different types. About the only thing they have
in common is that they are all used as flame retardants and all
contain bromine as active flame retarding mechanism. Other than
Deca-BDE, which is a general purpose FR and used in many different
applications, most BFRs have fairly narrow applications due to
their different chemical properties. Their different properties
in turn lead to differences in toxicology and environmental behavior.
As a result, we believe that each BFR should be evaluated and discussed
individually.
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